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Objective : To improve the therapeutic effect of inflammatory bowel disease ( IBD ).

目的: 进一步提高炎症性肠病的治疗效果.

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Osteoporosis in patients with inflammatory bowel disease: risk factors, prevention, and treatment.

骨质疏松症的炎症性肠病患者: 危险因素, 预防和治疗.

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Animal models inflammatory bowel disease can be induced by chemical, immunology, genetics and other access methods.

炎症性肠病动物模型可通过化学性诱导 、 免疫学 、 遗传学等方法获得.

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Early diagnosis and treatment is important for the patients with osteoporosis in inflammatory bowel disease.

对于炎症性肠病患者并发骨质疏松者应及早诊断及治疗,早期干预可减轻IBD患者骨质疏松的发生与发展.

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CONCLUSION: NF - κ B decoy ODNs will become a new gene drug for treating inflammatory bowel disease ( IBD ) .

结论: NF - κ BdecoyODNs 有望成为治疗IBD的一种新型基因药品.

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Chronic diarrhea is usually related to functional disorders such as bowel syndrome or inflammatory bowel disease.

而慢性腹泻往往和相应的肠胃功能紊乱有关,例如肠易激综合征或炎症性肠病.

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