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F - actin was labled and examined using fluorescence microscope.

标记F-actin,荧光显微镜下观察.

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Even , upregulation of MOMA - 2 and downregulation of SM - actin were also detected in latter ( P < 0.05 ).

MOMA -2 表达增加,SM-actin表达降低 ( P<0.05 ).

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Methods � � - actin pseudogenes were found out with bioinformatics databases such as EMBL, PSEUDOGENE.

方法利用EMBL 、 PSEUDOGENE等生物信息数据库,寻找β-actin的假基因.

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The gene expression of a - actin mRNA were determined by using the quantitative reverse transcription PCR technology.

利用半定量反转录聚合酶链式反应(QRT-PCR)法测定骨骼肌α-actin基因表达.

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RT - PCR was used to determine GK and PEPCKgene expression ( corrected by � � - actin ) .

用RT -PCR 检测GK和 PEPCK mRNA表达水平的变化.

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The role of actin filaments was disused in this paper.

本文讨论了肌动蛋白微丝骨架在细胞融合过程中的作用.

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Upon discontinuing dexamethasone, cross - linking of the actin networks was reversible.

当终止地塞米松的作用后,小梁细胞内的肌动蛋白交联是可逆的.

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And the chromosomes and organelles were successively connected with actin filaments during cytomixis.

在细胞融合过程中,有微丝骨架纤维与穿壁转移的染色质和细胞器相连.

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A large portion of the cytoskeleton consists of threadlike microfilaments composed mainly of the contractile actin.

细胞骨架大部分由微丝组成,微丝主要由可收缩的肌动蛋白组成.

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Cells are likely to be deteched when the quantity of actin filament decreases.

细胞中肌动蛋白纤维含量减少时,细胞易从基质上脱落.

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The Molecular Study of Nuclear Actin Involving in the Regulation of Gene Transcription.

细胞核肌动蛋白与RNA聚合酶关系的研究.

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Conclusion the decreased actin activity is one of the reasons the decline of cardiac systolic potential.

结论:肌动蛋白活性降低是引起收心肌收缩力降低的原因之一.

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In plant cells, actin cytoskeleton plays important roles in response to biotic and abiotic stresses.

植物细胞对外界生物或非生物的胁迫做出反应时, 微丝骨架起了重要的作用.

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Microtubules are shown in green, actin is in red and mitotic chromosomes are colored blue.

绿色为微丝, 红色为肌动蛋白,蓝色为染色质,中心点复制成两个.

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Disrupted actin cytoskeleton could be recovered by removal of the VD - toxin before the nucleoli were dyed.

在核仁能够看见以前,即核仁开始着色以前将毒素除去,可以使已经破坏的周质微丝骨架恢复过来.

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